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Breathing mechanics Abstract Breathing mechanics are the physical processes
that occur during inhalation and exhalation . In the inspiration , the use of
leads re spiratory muscles to an increase in intra thoracic volume, whereby in
the lung creating a negative pressure and air in the lungs is drawn. Due to the
elastic fibers of the lung parenchyma, the lungs passively contract when the
res piratory muscles relax, thus expelling the exhaled air ( expiration ).
To check the function of the lungs , the lung volumes can be determined.
Clinically, vital capacity is particularly important. It is defined as the
volume difference between maximum inhalation and exhalation . In the case of
so-
called restrictive lung diseases, the vital capacity is reduced. In contrast,
obstructive pulmonary diseases can be uncovered using the dynamic Tiffeneau
test: After maximum inspiration , the subject should exhale as much as possible
as quickly as possible.
The terms compliance and resistance describe how the lungs react to
deformations caused by breathing: A high level of compliance speaks for easy
mechanical defor mability of the lungs . A high resistance, on the other hand,
means that the air we breathe has to pass through a high flow resistance and
breathing is thus impeded.
•• Breathing process Breathing goal : Exchange of air in the lungs Phases of
breathing Breathing rest : Between two breaths, the in spiratory and ex piratory
forces are in balance, theintr apul monary pressure corresponds to the air
pressure. inspiration Ins piratory re spiratory muscles increase the volume of
the chest The lung skin ( pleura visceralis ) adheres to the inside of the
thorax ( pleura parietalis or pleura) via the liquid film in the pleural
space → lung volume is also increased As the volume of the lungs increases, the
intrapul monary pressure drops → negative pressure in the lungs Air follows the
pressure drop into the lungs → inspiration Expiration Reduction of the chest
volume through passive restoring forces of the lungs This reduces the volume of
the lungs → excess pressure in the lungs Air follows the pressure drop out of
the lungs → expiration Inspiration from : Expiration through:
res piratory muscles Diaphragm → breathing in the abdomen Mm inter costales
externi → breathing in the thorax Elastic restoring forces of the lungs (
passive) Ex piratory re spiratory muscles : Mm. inter costales interni
re spiratory muscles Mm sternocleidomastoid Mm serrati Mm pectorales Mm scaleni
Re spiratory auxiliary muscles : abdominal muscles Children mainly breathe in
the abdome nadults in the thorax !
will still be known to any older doctor as unsurpassed blessings. Orally, g-
strophanthin, which was only identified as an endogenous substance in 1991 (1),
has an outstanding effect in both the prophylaxis and treatment of heart attack
and angina pectoris, without showing any noteworthy side effects and without
being particularly expensive to be. Although the excellent therapeutic success
and the dynamic effects from 1950 to 2000 are documented with an overwhelming
abundance of (partly double-blind) studies and reports, g-strophanthin, the
"insulin of the cardiac patient",
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According to the manufacturer, a
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2. SENSATIONAL RESULTS From 1977 to 1987 there were a number of publications (
e.g. 2-3) on the world's best heart attack survival rates that a public
hospital in Berlin-West achieved with both oral g and intravenous k-
strophanthine, although previously Values were particularly bad due to the high
proportion of elderly people - only surpassed by a clinic in Sao Paolo, which
also worked with g-strophanthin.
The study of oral g-strophanthin in unstable angina pectoris in this clinic (4)
showed complete absence of symptoms after preventive use of gastro-resistant
capsules in 122 of 146 patients after one week and in 146 of 148 patients after
two weeks (98.6 percent) , with all other previous medications including side
effects omitted (ß-blockers, calcium antagonists, nitrates, etc.)
In 85 percent of the acutely admitted patients who received the strophanthin
for the first time, an effect was seen within 5-10 minutes (bite capsule with
perlingual absorption) (5). Patients could help themselves with oral g-
strophanthin before the emergency doctor arrived, as the following experiences
underline:
A German mine with oral g-strophanthin therapy underground no longer had a
single heart attack victim in 10 years, although previously there were an
Exits from the shaft due to angina pectoris and
heart attacks decreased by 80 percent. Strophanthin was not even given
preventively, but only in the case of an acute attack (6-7).
There are many more examples. The practical experience of more than 4,000
doctors documented in the 1980s and 1990s, who usually judge oral g-
strophanthin very positively, is instructive (10-11). Around 3,000 doctors in
The
homeopathic g-strophanthin (D4) is also astonishingly positive, albeit to a
lesser extent.
There are also a whole series of other pharmacodynamic studies (some of them
double-blind) in humans with significant improvements in pain symptoms and
performance, the ECG, high blood pressure, cardiac work, the flow behavior of
the blood and blood flow to the heart muscle through oral g- Strophanthin.
There are also numerous studies on animals and cell cultures, for example
guinea pigs can swim more than three times as long after oral g-strophanthin
administration (!); where g-strophanthin prevents heart enlargement in the
event of overexertion (12); In mice with sepsis, g-strophanthin leads to a
fourfold increase in the survival rate (13).
In acute heart failure, strophanthin iv was recommended as the fastest-acting
glycoside by the textbook until 1994 (55), today more digoxin and without a
pharmacological reason.
3. HARDLY ANY SIDE EFFECTS The side effects of strophantin can in rare cases be
irritation of the mucous membrane to inflammation of the tongue mucosa or
diarrhea, which can be easily remedied by reducing the dose. Permanent damage
has never been observed; there is no danger of overdosing and no
contraindications apart from the pronounced sinus bradycardia (57). G-
We need the following information to calculate your BMI (body mass index): Your
weight (kg) Your size (cm) Your BMI is
Theoretical basics of the BMI: The BMI is calculated from the body weight [kg]
divided by the square of the body size [m 2 ]. The formula is: BMI = body
weightin m2 . The unit of the BMI is therefore kg / m 2 .
This means that a person with a height of 160 cm and a body weight of 60 kg has
a BMI of 23.4 [60: (1.6 m) 2 = 23.4].
The "desirable" BMI depends on age. The following table shows BMI values for
different age groups:
Age BMI 19-24 years 19-24 25-34 years 20-25 35-44 years 21-26 45-54 years 22-27
55-64 years 23-28 > 64 years 24-29 BMI classi fication (according to DGE,
nutritional report 1992):
class ification m w Underweight <20 <19 Normal weight 20-25 19-24 Overweight 25-
30 24-30 Obesity 30-40 30-40 massive obesity > 40 > 40
.of the. ,of the,